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Regenerative Molecular Complex Cellular Metabolic Regulator
Revercell 2G
Ageing is a consequence, among other things, of an accumulation of unrepaired molecular damage that limits functions and gradually deforms the cell cycle. This damage occurs repeatedly and cumulatively, increasing to a lesser or greater degree depending on certain factors, such as genetics, lifestyle, nutrition, etc.
The Biocell Ultravital Bioresearch Institute is proud to incorporate into its portfolio of cell therapies REVERCELL 2G, a new product that acts mainly to minimise molecular damage, interacting with the mitochondria to extend proper functions in the life of our cell pattern.
Taking the example of scientist John Denu, who compares these cell processes to a production factory, with the passing of time, if the proper maintenance measures are not taken, everything will start to fail little by little. This occurs in cells due to the continuous effects of the accumulation of damage and mutations and the metabolic loss of functions that gradually make the life of our cell pattern deficient, causing it to degenerate as a result.
Poor cell function unleashes a chain of problems: the accumulation of bad proteins that cause part of the cell damage which, consequently, is reflected in tissue damage, organic damage and systemic damage. This is followed by the onslaught of diseases, many of them irreversible. It is for this reason that Biocell Ultravital’s focuses its research efforts on scientific development in the area of cell renewal and prevention.
During the past 30 years, regenerative research efforts focused progressively on counteracting or preventing the damage caused by so-called oxidative stress. This is generated by the action of Free Radicals, oxygen molecules missing an electron, produced by the mitochondria during ATP synthesis, in converting food into energy. In this regard, modest results were achieved with the use of high doses of so-called anti-oxidant vitamins: A, C, E, Selenium, Zinc, etc., an incredible phenomenon discovered at Cornell University going almost unnoticed.
Background Information:
Since 1934, with the restricted calorie experiments conducted on rats at Cornell University, reproduced consistently worldwide with different types of living creatures, from yeast to long-living mammals, it has been known that a diet very low in calories but rich in nutrients can extend animals’ lives by up to 60%. Until recently, this significant life extension was rightly attributed mainly to a lesser production of free radicals, due to reduced food intake; decreased levels of insulin, cholesterol and blood pressure, and a drop in body temperature, but this did not explain the reason why older animals subjected to a restricted calorie diet showed signs of rejuvenation, such as: Increased activity in general, including sexual activity, restored skin radiance, improved reflexes and learning ability.
Today, it is believed that restricted calorie diets, in addition to providing the aforementioned benefits, turn off or induce genetic silencing in damaged genes, separated from chromosomes, which begin to express and replicate large quantities of undesirable proteins, when they should be silent, which contributes strongly to ageing.
The severely reduced calorie intake imposed by a restricted calorie diet forces the body's cells to go into “survival mode,” resulting in the above benefits. These cease progressively in the presence of a normal diet. In other words, when a body takes in sufficient food, it does not need to remain in “survival mode.” Thus, it is evident that there is a strong connection between a person’s nutrition and his longevity.
It is deemed that the benefits of a restricted calorie diet can be extrapolated to human beings, who should not be an exception among all the animals researched. However, given man’s life span, several decades would be necessary to verify the results. This is why living organisms with short life spans are used, such as yeast, flies, rats, dogs and primates, etc., to conduct the bioresearch on this phenomenon. Moreover, it is highly unlikely that an individual could withstand for much time the rigours of a diet as low in calories as the one proposed.
A few years ago, the scientific community detected that caloric restriction causes the activation or expression of SIR (silent information regulator) genes, which contribute to preventing the production of large quantities of undesirable proteins, as a result of the DNA split off from chromosomes due to damage caused by oxidative stress, which would ultimately destroy the cells in question. It is estimated that some 8 human genetic silencing genes exist, known as SIRT1 through SIRT8. SIRT genes may rightly be called longevity genes.
More recently, it has been determined that chemical substances exist that are capable of expressing SIRT genes, in a manner similar to what happens in cases of caloric restriction, without the need to incur in this practice. This is known as caloric restriction mimetics. Included among these substances are: grape resveratrol, tea catechins, soy genistein, etc.
Biocell Ultravital’s research has been strongly geared towards the use of the most promising element on this list, in synergy with other components that contribute to optimising the action thereof.
The Biocell Ultravital Bioresearch Institute is proud to incorporate into its portfolio of cell therapies REVERCELL 2G, a new product that acts mainly to minimise molecular damage to the mitochondria, where harmful free radicals are produced during ATP synthesis, and damage to DNA’s ability to repair itself in cell nuclei, thus extending the normal functionality of our cell pattern.
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